Triplethought wrote: ↑Tue Jul 13, 2021 5:58 pm
Congrats. But Chinese stocks are makin me nervous these days. Maybe it's cuz the bastards gave me Covid
Some Chinese stocks will shoot to the moon, others will die on the vine.
MOMO is pretty much guaranteed to go up in value, purely because it kind of sounds like "Mo' Money" . . .
I suspect most are a little bit worn out about by the cv19 and vaccine debates, and this is speculation on my part combined with some science and knowledge, but what happened to you might be a manifestation of what is called "pathogenic priming," or "antibody-dependent enhancement," which really mean the same thing. Imo, it's starting to manifest . . .
I am tentatively guessing this fall/winter, it's going to be a terrible "common cold" and "flu" season. But it will all be blamed on the cv19 variants, I suspect, as well as those who are not vaccinated. Booster shots will be encouraged.
For those interested, here are a few links for your perusal, listed below in no particular order. There are also videos by very experienced physicians and scientists on this topic, but most have been removed by YT.
Anyways, here goes:
https://pubmed.ncbi.nlm.nih.gov/32908214/
https://pubmed.ncbi.nlm.nih.gov/32659783/
https://pubmed.ncbi.nlm.nih.gov/32507409/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7142689/
"The fact that pathogenic priming may be occurring involving autoimmunity against multiple proteins following CoV vaccination is consistent with other observations observed during autoimmunity, including the release of proinflammatory cytokines and cytokine storm. Similar to the SARS-CoV animal studies [6], found that mice vaccinated against MERS-CoV (Middle East Respiratory Syndrome) development exaggerated pulmonary immunopathology when challenged with the MERS virus following vaccination. They reported that lung mononuclear infiltrates were observed in all groups after virus challenge, and that increased infiltrates that contained eosinophils and the eosinophil promoting IL-5 and IL-13 cytokines were observed only in the vaccinated animals.
Pathogenic priming may be more or less severe in vaccine or infection induced immune responses to some proteins than for others due to original antigenic sin; the immunologic reaction against self-antigens may be made less severe as fast-evolving viruses evolve away from the original vaccine type. Thus, the screening of immunogenic epitopes for pathogenic priming potential via homology may be augmented by studies of autoantibodies that cross-react with epitopes included in vaccines."
Eh, don't shoot the messenger . . . these days, I am a quiet, thin-skinned, law-abiding Canadian citizen.
